GINGIVAL DISEASE
(GINGIVITIS AND PERIODONTITIS)
What is gingivitis and periodontitis?
- 
Gingivitis is the inflammation of the gingival tissues with no bone loss present. 
- 
Periodontitis is inflammation in the periodontist affecting gingival tissues, periodontal ligament, cementum, and support bone. 
​
(Wilkins, 2017, page 313)
Risk Factors for Periodontal Disease:
- 
Tobacco Use - 
More than 40% of cases of periodontitis are a result of cigarette smoking.​ 
 
- 
- 
Diabetes Mellitus - 
Periodontal disease and diabetes mellitus have a bidirectional​ relationship, meaning a patient who does not control blood glucose is more likely to have more severe periodontal disease. 
 
- 
- 
Psychosocial Factors - 
Individuals under stress are more likely to have alveolar bone loss.​ 
 
- 
- 
Medications - 
Specific systemic medications can lead to gingival enlargement and encourages plaque- induced biofilm retention.​ 
- 
Certain medications include Phenytoin, Cyclosporine, and Nifedipine. 
 
- 
- 
Nutritional Status - 
Poor diet that includes soft sugary foods can adhere to the ​teeth and tissues more than fibrous, firm foods. 
 
- 
- 
Genetic Predisposition - 
From 33% to 39% of the risks for periodontal disease is related to genetic factors. ​ 
 
- 
- 
Host Response - 
Alterations in the immune response may result in periodontal damage.​ 
- 
Overly active neutrophils may be responsible for the destruction seen in periodontal infection. 
 
- 
- 
Osteoporosis - 
Risk factors for osteoporosis are also risk factors for periodontitis, including smoking, nutritional dificeinces, glucocorticosteroid use, and immune dysfunction. ​ 
- 
Greater alveolar bone loss is common with osteoporosis. 
 
- 
- 
Other Systemic Conditions (such as Down syndrome) 
- 
Dental Factors - 
Congenital abnoramalities such as crowded or overlapped teeth could be a local contributing factor for periodontal disease. 
 
- 
- 
Poor Oral Home Care - 
Lack of proper oral home care can contribute to the accumulation of plaque - induced biofilm build up. ​ 
 
- 
(Wilkins, 2017, pages 313 - 316)

